My understanding is that albinism results from defective production of melanin from tyrosine. The metabiloc pathways are pretty complex. The original black rat albino had didn't produce the precursor tyrosine so the process was interrupted at that level. The T plus albino looked identical but did produce tyrosine. The defect was further along in the metobolic process but the end result was the same. The popular thinking is that defects that occur further along in the metobolic process might be "leaky". Its a good theory but to my knowledge (limited) nobody has done the requisite skin test to prove it.

Point is that some of the newer more typical albinos might also be T plus. Using T plus as a descriptor might be popular and serve a purpose but without the reaction test we can't say its factual.
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