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Posted by: Rainshadow at Tue Feb 28 09:41:36 2006 [ Email Message ] [ Show All Posts by Rainshadow ] 1.No, 2.Too early to tell.(however the first "patternless" Motley was produced from gene carrier to non-gene carrier,so although it doesn't make sense,it's a good question.) 3.No, 4.No.....The name "Harlequin" is not synonymous with a monotypical mutation,it is the name of the bloodline that produces anomolous results. I've always felt that in this way it is similar to the "Jungle",which is why so many people have had trouble understanding those also. it is much more important to identify the bloodline relationship,continuity,and,or parental status,than the appearence of any one individual produced from either of these lines. Even simple mutations do not always follow strict genetic calculations,so when dealing with "variably expressed" non-recessives you've got to do the long version of the equasion rather than try to hammer a square peg into a round hole,or jumping the gun in catagorizing the nature of the genetic behavior. I believe the Harlequin bloodline may end up providing us with an example of "incomplete dominance" (which is hard to prove,because I'm not aware of any clear,documented cases of it in boa constrictors.) hope this helps,(at least stimulate more thought! *lol*) [ Hide Replies ]
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