Hello Vinnie, I have never claimed to be a know it all...and on these forums, I mainly ask questions......I also am man enough to admit if i am wrong about something, and I take no offense to anyone opposing any of my views....





I just have read this forum and others and have...ideas...questions...and just think that if we are ever going to go anywhere in boas...breaking new ground is the answer...







Below is just some info that leads me to believe this...but as you will see without a DOPA test...it is impossible to know this for sure..but in my understanding just the fact that the Kahl and Sharp strain are incompatable...leads me to believe this.



Then another statement that I read says that there can be as many as 12 genes resonsible for formation of Melanin...



...here



The differentiation of melanophores and the formation of melanin may be controlled by at least 12 genes. However, genetic analyses of these color genes have not yet been completed.



Think about that in terms of the T positive morphs we already have... and Caramels, and Paradigm...All can, and maybe have this Sharp strain gene...which could be what was found with the Paradigms....but if it could be that 12 other genes could control or influence this...we may see many different morphs come from this....





http://www.bio.nagoya-u.ac.jp:8000/melanin.html



here is just a small exerpt from this...





The colorless melanophore remains in the state of incomplete pigmentation. The possible explanation of this feature are as follows; (1) colorless melanophores are deficient in substrate (tyrosine), (2) they contain an inactive enzyme precursor, (3) in these cells, intermediate products in the processes of the formation of melanin are carried off to other metabolic pathways. It is difficult to analyze the control mechanisms which play a role in skins of the various color types. The activities of dopa oxidase and tyrosinase in the skin are higher in the orange-red and white types than in the brown and gray types. This fact is quite contrary to the first expectation. The results indicate that the activity of tyrosinase is very low when melanin is fully formed, and the action of gene B is restricted only to the stage during the time of formation of melanin. As granules having tyrosinase activity become more fully melanized, perhaps tanning in a chemical sense occurs. Some of active sites on the enzyme may be blocked so that apparent activity of the enzyme is no longer noticed in more fully melanized melanophores



Thanks Andy Federico
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