Like amelanism and anerythrism, leucism may potentially occur in any species of animal. It is a much rarer mutation, but it certainly isn't specific to Texas rats alone. I think it is quite possible that it may have originated as a spontaneous mutation in a pure black rat. Proving it, one way or the other, is something that would be very hard to do. Possibly DNA analysis could provide some clues, but maybe not a definitive answer.
Assuming, for the sake of argument, that it didn't spontaneously arise in blacks, then one has to believe that it is a lindheimeri intergrade. If this is the case, then one has to ask, does it really matter? We know that there are many places where obsoletus subspecies naturally intergrade, and we don't get terribly hung up on that fact when we're dealing with WC animals, so why is it such a big deal when the animals are CB?
Suppose then, that some time in the past somebody mated a LTR to a black, then mated their offspring together to produce a clutch of 50% intergrades in which 1:3 of the babies were leucy. Taking one of those leucy babies and breeding it to a pure black, the resulting clutch consisted of babies that were all 75% pure blacks, and all het for leucy. Breeding two of them togetehr, again yielded a clutch of 1:3 leucistics, but these were 75% pure black. Repeating that process, the third result would be leucys that were 87% pure black, the fourth would be babies 94% pure, and the fifth 97%. At 97% purity, I think that it no longer matters where the leucy gene originated, because 97% purity is probably as high as it gets in a lot of the subspecies foundation stock anyhow (BTW, I estimate that it would take about 30 years to achieve this level, allowing three years per generation). Just my POV, and probably not worth much.
As Dwight has stated, this topic has been well covered in the past.
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tricia