"That is an interesting situation.... It seems to lean towards the possibility that the pied gene could be expressed at more than one gene location. If one location is the expression, a marker belly is visible in the hets.... if the other allele is the expression location, then no belly stripes are present in the hets."
Are you talking about the same gene getting moved around on the chromosome? I heard about something like that but I think it was some special situation and not a normal thing to happen.
Or maybe the marker expression or lack there of is due to another gene location (i.e. a different gene altogether) that is sort of a pied helper gene?
"Another possibilty could be that the stripe belly is an independant gene that just happens to be more common in the pied line, maybe due to restricted gene pool?"
I think pastel is the only other mutation that has been outbred as much as piebald judging by all the possible hets seen so far (spider and other dominant types probably aren’t far behind). Piebald has probably been outbred for the same reasons as the consistently dominant type mutations - visible hets. At least the sporadic visible hets are an encouraging factor on top of the motivation of a high dollar morph that you want to be as strong as possible.
The way the belly marker has followed down into some of the 50% hets and down to half of their 25% het offspring it must either be the pied gene it's self or a very closely linked gene. Still doesn't explain the known het lines that don't have the marker.