The size of lion/tiger offspring depends on which species makes up which sex and is due to genetic imprinting. Certain traits are more important to each species and sex, and therefore, females have different genes turned on/off than the males do.
So ligers are male lion and female tiger crosses.
Lion breeding is very competitive and it is in the males best interest to produce large young in order to ensure his genes are passed on. The females are interested in producing as many young as possible and are not worried about the size. Therefore, the males pass on genes with growth turned on, while the females growth genes are inactivated.
Tigers are solitary and do not have the same type of competitive breeding. Males have no need to increase the size of offspring, and females have no need to restrict these increases.
When you match the male lion (genes for growth) with the female tiger (no restriction) you get the very large liger.
Male tigers (normal growth genes) with female lions (restricted growth) produce the much smaller tigon.
Interestingly, this can also lead to problems in humans. When the nonimprinted (active) allele is inactivated by deletion or mutation, the imprinted (inactive) allele is unable to provide normal biologic activity. If the male copy of an allele on chromosome 15 is deleted, the child is born with Prader-Willi syndrome. If the female copy of the same allele is deleted, the child is born with Angelman syndrome.
Slowed growth in patternless atrox could be a result of some of the genes dictating the pattern, lying in close approximation to a gene for growth on the same chromosome. A deletion or mutation could inactivate that section of the chromosome. Maybe they are on different chromosomes and there is a recombination that disrupts both loci. I would think in situations like these, there would be other problems present as well though. I sure don't have the answer, but I don't think this is due to genetic imprinting.
Jason